Neurofibril example sentences
neu·ro·fi·bril
noun anatomy
neurofibril (noun) · neurofibrils (plural noun)
- a fibril in the cytoplasm of a nerve cell, visible by light microscopy.
"Neurofibril" Example Sentences
1. The researchers observed significant neurofibril tangles in the brain tissue.2. Neurofibril degeneration is a hallmark of several neurological diseases.
3. Damage to neurofibrils can impair neuronal function.
4. The study focused on the role of tau protein in neurofibril formation.
5. Abnormal neurofibril accumulation contributes to cognitive decline.
6. Microscopic examination revealed numerous disrupted neurofibrils.
7. He presented his findings on the relationship between amyloid plaques and neurofibrils.
8. Further research is needed to understand the precise mechanisms of neurofibril pathology.
9. The neurofibril network plays a crucial role in maintaining neuronal structure.
10. Disruption of neurofibrils can lead to neuronal death.
11. She dedicated her career to studying neurofibril dynamics in Alzheimer's disease.
12. The drug's efficacy was evaluated based on its impact on neurofibril density.
13. Neurofibril pathology is a complex process involving multiple factors.
14. Advanced imaging techniques are used to visualize neurofibrils in the brain.
15. The clinical presentation often reflects the severity of neurofibril degeneration.
16. They hypothesized that oxidative stress contributes to neurofibril damage.
17. This innovative therapy targets the underlying mechanisms of neurofibril dysfunction.
18. Neurofibril lesions are commonly observed in post-mortem brain examinations.
19. Understanding neurofibril structure is essential for developing effective treatments.
20. The presence of numerous neurofibrils indicated advanced disease progression.
21. His research focused on the genetic factors that influence neurofibril aggregation.
22. The team developed a novel method for quantifying neurofibrils in brain samples.
23. Early detection of neurofibril changes could lead to improved outcomes.
24. Neurofibril disruption is a significant contributor to synaptic dysfunction.
25. The article highlighted recent advances in neurofibril research.
26. Chronic inflammation may accelerate neurofibril degeneration.
27. They examined the correlation between neurofibril burden and cognitive performance.
28. The study showed a strong association between neurofibril density and disease severity.
29. Neurofibril pathology is a central feature of many neurodegenerative diseases.
30. Improved understanding of neurofibril biology could revolutionize therapeutic strategies.
31. Are neurofibrils solely responsible for the observed neurological deficits?
32. The degradation of neurofibrils was observed using advanced microscopy.
33. What is the precise role of tau in neurofibril formation and aggregation?
34. How do genetic mutations affect neurofibril stability?
35. The impact of environmental factors on neurofibril health is still under investigation.
36. The experiment aimed to assess the effects of the drug on neurofibril morphology.
37. Neurofibril research holds great promise for the development of novel therapeutics.
38. This review article summarized current knowledge of neurofibril structure and function.
39. Further studies are needed to clarify the exact role of neurofibrils in disease pathogenesis.
40. The findings suggest a potential therapeutic target within neurofibrils themselves.
41. Investigating neurofibril dynamics is key to understanding disease progression.
42. Dysfunctional neurofibrils contribute to the loss of neuronal connectivity.
43. The clinical trial evaluated a new drug designed to stabilize neurofibrils.
44. We observed a significant reduction in neurofibril pathology following treatment.
45. Neurofibril dysfunction is often accompanied by widespread synaptic loss.
46. The researchers developed a new model to study neurofibril aggregation in vitro.
47. Long-term studies are needed to assess the long-term effects on neurofibrils.
48. Neurofibril alterations can be detected even in the early stages of disease.
49. Targeting neurofibrils might be a promising strategy for disease modification.
50. The study's results shed new light on the role of neurofibrils in neurodegeneration.
Common Phases
1. Neurofibrillary tangles are a hallmark of Alzheimer's disease.2. The accumulation of neurofibrillary tangles correlates with cognitive decline.
3. Neurofibrillary tangle density is a significant pathological marker.
4. Studies investigate the role of tau protein in neurofibrillary tangle formation.
5. Neurofibrillary tangle pathology is progressive in nature.
6. The presence of neurofibrillary tangles indicates neuronal damage.
7. Researchers are exploring therapeutic strategies targeting neurofibrillary tangles.
8. Imaging techniques can visualize the distribution of neurofibrillary tangles.
9. Neurofibrillary tangle burden varies between individuals with Alzheimer's disease.
10. The pathogenesis of neurofibrillary tangle formation remains incompletely understood.
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