Synapses example sentences

"Synapses" Example Sentences

1. The neurons communicated across synaptic connections.
2. The growth of new synapses strengthens the connections between neurons.
3. Synaptic signaling transmits information between cells in the nervous system.
4. Memories are formed by the strengthening of synapses between neurons.
5. Repeated activation leads to the formation of more synapses between neurons.
6. Neurotransmitters are released at the synapses to transmit signals.
7. Learning and memory involve changes in the strength of synapses.
8. Dendrites receive signals from the axons at synapses.
9. The brain has trillions of synapses that allow neurons to communicate.
10. Synaptic plasticity refers to the ability of synapses to change and adapt.
11. Neurotransmitters bind to receptors at the synaptic cleft.
12. Dopamine is a neurotransmitter involved in regulating synaptic activity.
13. Synaptic vesicles contain neurotransmitters that are released at synapses.
14. Drugs can impact synaptic transmission by altering neurotransmitters.
15. Synaptic activity produces electrical signals that are transmitted through neural circuits.
16. Neurodegenerative diseases can lead to the loss of synapses in the brain.
17. Certain medications may help promote the growth of new synapses.
18. Electrical synapses allow neurons to communicate through direct connections.
19. Chemical synapses transmit signals via the release of neurotransmitters.
20. Scientists are studying how to grow and rewire synapses to repair brain damage.
21. Brain injuries often involve damage to synapses that disrupt neural pathways.
22. Synaptic fatigue can occur when neurotransmitters are depleted after excessive activity.
23. Mental stimulation may help preserve existing synapses and even promote the growth of new ones.
24. Axons transmit signals to dendrites across the small gap known as the synaptic cleft.
25. Synapses undergo constant change through processes of reinforcement and elimination.
26. New experiences can prompt the brain to make new synaptic connections.
27. Synaptic dysfunction may play a role in conditions like Alzheimer's and depression.
28. Synaptic pruning in adolescence involves the elimination of unused synaptic connections.
29. Certain diseases involve the loss of synaptic insulation known as myelin sheaths.
30. The function and number of synapses often decline with age in the typical aging brain.
31. Neurotransmitters bind to both pre- and post-synaptic receptors across the synaptic cleft.
32. Medications that inhibit synaptic activity may help control conditions like epilepsy and spasticity.
33. During development, the brain forms an excess of synapses that are later pruned back.
34. Synaptic signaling occurs in microseconds to enable the high-speed functioning of the nervous system.
35. Neurotransmitters must rapidly diffuse across the synaptic cleft to activate post-synaptic receptors.
36. Severing axons can disrupt synaptic signaling and lead to denervation of target cells.
37. Stimulating environments may promote activity-dependent synaptic plasticity in the brain.
38. Brain injuries often involve damage to axons that prevents them from forming new synapses.
39. Drugs of abuse can drastically impact synaptic signaling by flooding the synaptic cleft.
40. Inhibiting excess synaptic activity may help reduce neuronal excitotoxicity in certain conditions.
41. The synapse is the basic signaling unit that enables information processing and storage in the brain.
42. Astrocytes play an important role in regulating synaptic activity and formation in the central nervous system.
43. Microglia prune weak synapses during development and help eliminate damaged synapses following injury.
44. Magnetic stimulation can noninvasively impact synaptic activity within targeted neural circuits.
45. Action potentials in the axon trigger neurotransmitter release from vesicles at the synaptic terminal.
46. Antidepressants may increase synaptic activity by enhancing levels of neurotransmitters like serotonin and dopamine.
47. Drugs that block glutamate receptors can help protect against excitotoxicity caused by excess synaptic signaling.
48. Neurotrophic factors promote synaptic plasticity by stimulating the growth and reinforcement of synapses.
49. Neurotransmitters bind to both ionotropic and metabotropic receptors located on the post-synaptic membrane.
50. Microglia engulf and digest damaged synaptic elements to stimulate regrowth following nerve injury.
51. Electrical synapses allow for the rapid and direct transfer of signals between neurons.
52. Synaptic efficacy refers to the ability of a synapse to transmit signals between neurons.
53. Synaptic vesicles store, transport and release neurotransmitters into the synaptic cleft.
54. Calcium signaling within dendritic spines regulates the formation and plasticity of synapses.
55. Neurotransmitters are rapidly taken back up by pre-synaptic neurons or broken down by enzymes.
56. Synaptic activity produces miniature excitatory or inhibitory post-synaptic currents.
57. Synaptic tagging captures plasticity-related proteins to strengthen newly formed synapses.
58. Decreased synaptic activity leads to reduced release of neurotrophic factors in some neurons.
59. Noxious stimuli can cause long-term potentiation of synapses involved in processing pain.
60. Electrical stimulation can be used to induce plasticity changes within targeted synaptic pathways.

Common Phases

1. Neurons communicate with each other through synapses.
2. Synapses allow electrical and chemical signals to pass from one neuron to another.
3. Synaptic connections are formed and strengthened when neurons fire at the same time.
4. Learning and memory involved altering synaptic connections between neurons.
5. The brain contains trillions of synapses that transmit electrochemical signals between neurons.
6. Synapses are formed when the axon terminal of one neuron is close enough to the dendrite or cell body of another neuron.
7. Neurotransmitters are released at the synaptic cleft between the neurons to transmit the signals across the synapse.
8. When we learn new information, our brains form new synapses and strengthen existing synaptic connections.
9. Long-term potentiation occurs when synaptic connections are permanently strengthened by repeat firing.
10. Synaptic pruning occurs during childhood where unused synaptic connections are eliminated.
11. Strong synaptic circuits form the basis of our memories and skills.
12. Damage to synapses can lead to neurological disorders and cognitive decline.
13. Synaptic plasticity refers to the ability of synapses to change and remodel in response to experiences.
14. Synaptic dysfunction is associated with many serious mental and neurological conditions.
15. New synapses continue to form in adults, particularly when learning new skills.
16. Alzheimer's disease involves deterioration of synapses in regions of the brain involved with memory and cognition.
17. Exercise helps promote the growth of new synapses and aids in synaptic plasticity.
18. Neurotransmitters modulate synaptic activity by binding to receptors on the postsynaptic neuron.
19. The synapse is the basic functional unit of information transfer in the nervous system.
20. Certain medications work by targeting receptors and signaling at synapses.
21. As we age, many of our synapses naturally deteriorate resulting in cognitive changes.
22. When we first learn something new, the associated synapses are weak and unstable.
23. Medications that enhance synaptic signaling have shown promise in treating cognitive disorders.
24. A neuron has thousands to tens of thousands of synapses on its dendrites and cell body.
25. Many psychiatric medications work by altering activity and signaling at synapses.
26. Medications that enhance synaptic plasticity may help maintain cognitive abilities in older adults.
27. Drugs of abuse can alter synaptic signaling patterns and damage synaptic structures.
28. Psychological stress can weaken synaptic connectivity and impair cognitive function.
29. Early life experiences shape how synapses form and strengthen in the developing brain.
30. Neurotrophins help strengthen synaptic connections and promote neuronal survival.
31. Learning helps wire neurons together by forming and reinforcing synapses.
32. Synaptic vesicles contain neurotransmitters that are released at the synapse.
33. Neuromodulators help regulate synaptic signaling and plasticity.
34. Synapses in certain regions of the brain seem more susceptible to age-related deterioration.
35. Weakened synapses are thought to underlie memory loss in conditions like Alzheimer's.
36. Neural circuits are formed by interconnected networks of synapses between neurons.
37. Repeated stimulation of synapses leads to long-term potentiation and synaptic strengthening.
38. Certain nutrients and hormones support synaptic health and function.
39. Synaptic activity triggers a signaling cascade that results in synaptic plasticity.
40. Synaptic dysfunction occurs in many neurodegenerative diseases of aging.
41. As we mature, experience strengthens the synapses most used and weakens underused ones.
42. Synaptic targets for drugs include neurotransmitter receptors and ion channels.
43. Synaptic terminals contain mitochondria that provide energy for neurotransmitter release.
44. Fixing synaptic dysfunction may be an effective strategy for treating cognitive disorders.
45. Imaging technologies are revealing new insights into synaptic functioning in the living brain.
46. Synapses exhibited a high degree of molecular specificity and structural organization.
47. Synaptic levels of certain chemicals are altered in conditions like depression and anxiety.
48. Neuroinflammation can damage and weaken synapses, impairing cognitive performance.
49. Neurotrophic factors help promote synaptic development and maintenance.
50. Synapse formation and pruning underlies the brain's developmental reorganization.
51. Synaptic signaling is highly dynamic, changing moment to moment based on experience.
52. Most pharmacological treatments aim to enhance synaptic transmission in some way.
53. Synaptic vesicles contain the molecular machinery for neurotransmitter release.
54. Synapses are the brain's primary information processing units.
55. Damage to synapses is an early change seen in Alzheimer's disease.
56. Neurogenesis can form new synapses that integrate into existing neuronal circuits.
57. Astrocytes help regulate synaptic formation, transmission and plasticity.
58. Magnetic stimulation techniques can be used to stimulate synaptic activity non-invasively.
59. Spines on dendrites are sites of most excitatory synaptic connections in the brain.
60. Imaging techniques are revealing new insights into structural plasticity at synapses.