Synapse example sentences

"Synapse" Example Sentences

1. The synapse transmitted the electrical signal from one neuron to another.
2. Synaptic transmission involves the release of neurotransmitters at the synapse.
3. The synaptic cleft is the small space between the axon terminal of one neuron and the dendrite of the next neuron at a synapse.
4. Synaptic plasticity allows synapses to strengthen or weaken over time.
5. Long-term potentiation is a form of synaptic plasticity that increases the strength of connections between neurons.
6. Dopamine is a neurotransmitter involved in synaptic transmission at many synapses in the brain.
7. Glutamate is the most common neurotransmitter used for synaptic transmission in the brain.
8. An action potential in an axon terminal triggers the release of neurotransmitters into the synaptic cleft.
9. Neurotransmitters bind to receptors on the postsynaptic neuron at the synapse.
10. The binding of neurotransmitters to postsynaptic receptors alters the postsynaptic membrane potential.
11. The neurotransmitter acetylcholine is used at many synapses, especially those involved in motor control and memory.
12. GABA is an inhibitory neurotransmitter that reduces the likelihood of an action potential at synapses in the brain.
13. Neurotransmitter receptors are located on the postsynaptic membrane at the synapse.
14. Excitatory neurotransmitters tend to make the postsynaptic neuron more likely to fire an action potential.
15. Inhibitory neurotransmitters make the postsynaptic neuron less likely to fire an action potential.
16. The presynaptic neuron releases neurotransmitters into the synaptic cleft between the presynaptic and postsynaptic neurons.
17. Signal transmission across the synapse is the basis for information processing and communication between neurons.
18. Synaptic transmission is essential for functions such as learning, memory, and motor control.
19. Alzheimer's disease is associated with synapse loss and dysfunction.
20. Neurotransmitters are quickly removed from the synaptic cleft after transmission by active reuptake or enzymatic breakdown.
21. Many drugs work by altering synaptic transmission in the brain.
22. Brain injuries can damage or destroy synapses.
23. Neurotrophic factors help regulate synapse formation and strength.
24. The number and strength of synapses in a neural circuit determine its capabilities.
25. Neuromodulators alter the strength and effectiveness of synaptic transmission.
26. Synapses within neural circuits undergo continuous modifications throughout life.
27. Neurotransmitter vesicles contain neurotransmitters that are released at the synapse.
28. Neurotransmitter reuptake involves transporting neurotransmitters back into the presynaptic neuron.
29. Certain diseases are associated with abnormal synaptic function.
30. Calcium ions play an important role in neurotransmitter release at the synapse.
31. Electrical signaling occurs across the synapse via ions flowing through ion channels.
32. Electrical synapses allow direct electrical coupling between connected neurons.
33. Chemical synapses utilize neurotransmitters for signaling between neurons.
34. Synaptic vesicles fuse with the presynaptic membrane to release neurotransmitters.
35. Neurotransmitters bind to metabotropic receptors that trigger intracellular signaling cascades.
36. Ionotropic receptors directly gate ion channels when neurotransmitters bind.
37. Retrograde messengers are released by the postsynaptic neuron to modulate presynaptic function.
38. Communication between neurons depends on the precise timing of synaptic transmission.
39. Neural networks are shaped by complex patterns of synaptic connections.
40. Synapses are continually remodeled throughout life in response to experience and activity.
41. Drugs that target synaptic function are used to treat a variety of neurological and psychiatric disorders.
42. Synaptic vesicles actively recycle neurotransmitters for reuse.
43. Microglia help prune excess synapses during development and in response to injury or disease.
44. Astrocytes provide support and regulation of synaptic function in the brain.
45. Excitatory and inhibitory synaptic balance is crucial for proper brain function.
46. Staining synaptic proteins allows visualization of synapses under a microscope.
47. Synaptic dysfunction is evident in many neurodegenerative disorders.
48. Imaging techniques allow in vivo studies of synapse formation and plasticity.
49. Dendritic spines are protrusions on the dendrites of neurons where many excitatory synapses form.
50. Synaptogenesis is the formation of synapses between neurons.
51. Synaptic dysfunction may underlie memory loss in Alzheimer's disease.
52. Neurotrophins help regulate synapse formation, stabilization and plasticity.
53. Retrograde messengers released by postsynaptic neurons regulate presynaptic neurotransmitter release.
54. Certain genetic disorders involve mutations that impact synaptic function.
55. Neuromodulators alter the strength of existing synapses within neural circuits.
56. Synaptogenesis occurs primarily during early development but continues into adulthood.
57. Synaptic inhibition reduces the excitability of the postsynaptic neuron.
58. Excitotoxicity occurs when excess glutamate overstimulates neurons, damaging synapses.
59. Neurotransmitter receptors are targeted by drugs that aim to modulate synaptic transmission.
60. Synaptic strengths are balanced in neural circuits to produce optimal responses and behaviors.

Common Phrases

1. The synaptic signal was transmitted across the gap.
2. The neurotransmitters helped facilitate communication across the synapse.
3. Synaptic plasticity leads to changes in neuronal connections.
4. The synapse allows the neuron to pass signals to other cells.
5. The axon terminal releases neurotransmitters at the synapse.
6. Action potentials are converted into chemical signals at the synapse.
7. Synaptic vesicles contain neurotransmitters that are released at the synapse.
8. Electrical signals are converted into chemical signals at the synapse.
9. Learning and memory formation involve changes at the synaptic level.
10. The presynaptic neuron released neurotransmitters into the synapse.
11. Synaptic transmission is key to neural communication and function.
12. Neurotransmitters bind to receptors on the postsynaptic neuron at the synapse.
13. Many synapses exist within the human brain and nervous system.
14. The synaptic cleft separates the presynaptic and postsynaptic membranes.
15. Long-term potentiation results in strengthening of synaptic connections.
16. Synaptic pruning occurs during brain development and maturation.
17. Synapses allow for both excitatory and inhibitory neural communication.
18. Synaptic dysfunction has been implicated in various neurological disorders.
19. Drugs that affect synaptic transmission can alter neural activity.
20. The speed of synaptic transmission depends on the type of neurotransmitter.
21. Synaptic fatigue occurs when neurotransmitters are depleted.
22. The synapse acted as a junction between the pre and postsynaptic neurons.
23. The synaptic terminals released a flood of neurotransmitters.
24. Synaptic activity was recorded using electrophysiological techniques.
25. Neurotransmitter receptors clustered at the postsynaptic density of the synapse.
26. Learning causes changes in the strength of synaptic connections.
27. Damage to synapses can impair neural communication.
28. Synapse formation occurs during neural development and growth.
29. Synaptic transmission failed due to the lack of available neurotransmitters.
30. Neurotrophins help regulate synapse formation, maintenance and plasticity.
31. Glial cells play an important role in regulating synaptic activity and transmission.
32. Synaptic vesicles fuse with the presynaptic membrane to release neurotransmitters.
33. The synapse allowed bidirectional communication between the two neurons.
34. New synapses formed as the neurons grew closer together.
35. The synapse malfunctioned, impairing communication between the neurons.
36. Synaptic cleft width affects how effectively neurotransmitters bind to receptors.
37. Synaptic degradation occurs as we age and contributes to cognitive decline.
38. Neurotransmitter levels affected the strength of synaptic transmission.
39. The synapse was activated, triggering an action potential in the postsynaptic neuron.
40. Synaptic integrity is essential for normal cognitive and motor function.
41. Synaptic vesicles dock and fuse with the presynaptic membrane in a complex process.
42. Neural circuits depend on precise connections between thousands of synapses.
43. Synaptic vesicles recycled neurotransmitters for reuse at the synapse.
44. Synaptic connections in the brain are remodeled throughout life.
45. Synaptic plasticity underlies learning and memory formation.
46. Neurons formed synaptic connections with each other through synaptic remodeling.
47. Glutamate is the major neurotransmitter at excitatory synapses in the brain.
48. The synapse failed to fire, silencing the postsynaptic neuron.
49. Neurotransmitters bound to receptors and triggered an action potential at the synapse.
50. Synaptic terminals branched out to form connections with multiple neurons.
51. Synaptic vesicles stored and released acetylcholine at the neuromuscular junction.
52. Acetylcholinesterase breaks down acetylcholine in the synaptic cleft.
53. Neurotransmitter reuptake helps terminate synaptic transmission.
54. Neurotransmitter spillover can activate extrasynaptic receptors at neighboring synapses.
55. Synaptic transmission failed due to damage to the presynaptic neuron.
56. Neural circuits rely on precise timing of synaptic transmission between neurons.
57. Neurotransmitters quickly diffused across the narrow synaptic cleft.
58. Synaptic dysfunction has been implicated in Alzheimer's disease and other disorders.
59. Neurotransmitter levels influenced the strength and efficacy of synaptic transmission.
60. The synapses connected regions of the brain involved in memory and emotion.